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National Institutes of Health, Bethesda, MD) and bred at the University of California, Los Angeles (UCLA).

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Human full-length HO-1 cDNA was amplified by polymerase chain reaction, and subcloned into the GV230 vector to construct recombinant HO-1 expression plasmids GC230-HO-1.

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Additional 5% acetic acid is applied to the areas being examined during the procedure, and the most abnormal acetowhite areas that are suspicious for AIN are biopsied with Baby Tischler Forceps.

The bacteria infect the hair follicles and cause red bumps and pus or pimple like bumps to develop.

Call your doctor right away if you have a skin reaction that bothers you or if the reaction makes it hard to keep putting this drug on.

The principal pharmacologic effect is augmentation of both innate and adaptive immune responses.

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As a result of these initial findings, a larger randomized, double-blind placebo controlled study was undertaken by Wolf et al.

Exclusion criteria includes conditions in the facial area that might impair evaluation, pregnancy or lactation, chemical or alcohol dependency, and allergy to imiquimod or study cream excipients.

Complete clearance of all AKs (including lesions revealed during treatment) was achieved in 36 percent of patients as compared to only 6 percent of those on placebo.

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Significant heterogeneity in pretreatment tumor microenvironments was observed, which illustrates that baseline immune profile is likely relevant to respond to immunomodulatory therapy.

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These results demonstrate that the TLR7 agonists could enhance the expression of costimulatory molecules and IL-12 by macrophages and DCs, potentially further promoting the activation and function of T and NK cells.

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IL-17 has a similar strong effect, almost completely abolishing skin disease (Fig.

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